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The ascitic fluid total protein concentration was greater than or equal to 2. .
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1 g/dL) and total protein concentration greater than 2. 5 is most consistent with exudative effusion.
Based on the SAAG of 0.
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The serum-to-ascites albumin gradient ( SAAG) accurately identifies the presence of portal hypertension and is more useful than the protein-based exudate/transudate concept ( and and ). High SAAG, low protein (HSLP) ascites, seen in 6.
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Graphics. In this group, 21/69 (30%) subjects in this group had received an agent (azathioprine, oxaliplatin, trastuzumab, or emtansine) that has been associated with development of non-cirrhotic portal hypertension. .
5, and 3. Patients with SAAG ≥ 1. 5 g/dL. . The formula is below. 5 g/dL Cancer, Tuberculosis, Chylous ascites, Pancreatitis Right HF, Budd-Chiari (early), veno-occlusive disease.
The ascitic fluid total protein concentration was greater than or equal to 2.
5 g/dL) occurs in the following 2 situations: (1) peritoneal malignancy or tuberculous peritonitis, which result in leakage of high-protein mesenteric lymph from an inflamed peritoneal membrane or obliterated lymphatics and (2) postsinusoidal or posthepatic causes of portal hypertension, in which the. Meanwhile heat oil in a pan and add the cumin seeds.
Serum:Ascitic Albumin Gradient (SAAG) = serum albumin – ascitic fluid albumin.
< 11g/L = low SAAG = exudate.
In this group, 21/69 (30%) subjects in this group had received an agent (azathioprine, oxaliplatin, trastuzumab, or emtansine) that has been associated with development of non-cirrhotic portal hypertension.
We herein report a case of a 42-year-old.
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